Renal anemia is common in patients with renal failure complications, anemia will not only affect the normal life of the patient to the patient much pain, but will exacerbate kidney damage. Our experts will be below renal anemia may make finishing, renal failure patients following areas can be made to prevent and manage, relieve symptoms of anemia.
Common cause anemia causes:
① uremic toxins damage to blood cells, red blood cells shortened life expectancy.
② erythrocyte plasma exists in inhibiting the growth factor. Uremic plasma to extract such isolated erythrocyte growth inhibitory factor, and with erythroid colonies in vitro method for identifying a molecular weight of about between 24000-94000.
③ erythropoietin (EPO) absolute and relative lack of:
The average normal human plasma EPO is 10-20mu / m / general anemia of up to 100 times normal or more. Chronic renal failure, a great part of nephron damage, EPO produces significantly reduced.
④ uremic patients on chronic hypoalbuminemia, blood raw materials shortage, such as: iron, folic acid, B12 deficiency is the cause of anemia.
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When ⑤ uremia bleeding tendency, within a few leftover blood dialyzer long-term dialysis, chronic blood loss also factors dialysate improper compatibility (sodium high temperature) in patients with hemolytic Dengjun, aggravated anemia.
⑥ lax dialysis treatment, the aluminum content is too high, or to correct a high phosphorus and application of aluminum and the like, can lead to overloading of aluminum, even aluminum poisoning. Aluminum can be combined with transferrin, it interferes with the binding of iron and hemoglobin, affect the synthesis of hemoglobin; the role of certain enzymes aluminum may also inhibit the synthesis of hemoglobin, and the impact of anemia. Features aluminum poisoning caused by anemia is a small cell hypochromic anemia.
⑦ secondary hyperparathyroidism: Impact PTH anemia can be: A, induce renal osteodystrophy, fibrous osteitis myelofibrosis, the number of red blood cells so that ancestor. B, PTH is the molecule toxins, red blood cells in bone marrow progenitor has a direct toxic effect, inhibition of erythrocyte calcium pump activity, so that calcium in erythrocytes increased fragility cells shortened life expectancy. C, reduce the reactivity of erythropoietin.
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